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The herbicide norflurazon (Predict) causes the new foliage to turn
white because it inhibits the production of carotenoids, which leads
to the destruction of the green chlorophyll in plants. Plants die
because there is no production of plant food in the leaves.
Norflurazon is applied preemergent and has limited soil mobility.
It is considered a nonselective herbicide but does not leach deep
enough in the soil to damage deeper-rooted established plants.
The herbicides diquat (Reward) and glufosinate (Finale) disrupt cell
membranes. They are nonselective, contact herbicides. Diquat injury
may be visible a few hours after application. Chlorosis (yellowing
resulting from the loss of green pigment) occurs within 2-5 days for
glufosinate. Good coverage is required. Plants can recover from
incomplete spray coverage.
Herbicides that prevent seedling root development [oryzalin (Surflan),
pendimethalin (Pendulum), prodiamine (Endurance)] stop the growth of roots
and shoots of germinating seeds or small seedlings by disrupting cell
division. Because they translocate to only a limited degree in plants,
they seldom control established weeds. They are highly selective for
grass control and usually do not leach in soils. Effective control
depends on sufficient rainfall soon after application or soil incorporation.
Fosamine (Krenite) inhibits bud development when applied to woody
plants late in the growing season, but before leaves start changing colors.
Except for pines, there is little or no visible effect (brownout) until
the following spring, when susceptible woody plants do not resume growth.
Since fosamine moves only from the leaves to the buds, uniform coverage
of the plant is necessary. Fosamine has no soil activity and does not
injure grasses at normal use rates.
A common characteristic of grass specific herbicides [fenoxaprop-ethyl
(Horizon), fluazifop-p-butyl (Fusilade), sethoxydim (Poast)] is that their
activity is limited almost entirely to postemergent control of annual and
perennial grasses. Nearly all non-grass species are resistant. These
herbicides are absorbed by the leaves and have little soil activity.
They are most effective when applied to unstressed, rapidly growing grasses.
Death of susceptible species takes a week or more. Crop oil concentrates,
nonionic surfactants, and nitrogen solutions increase their effectiveness.
Antagonism has been observed when these products are tank mixed with
auxin/growth regulator herbicides, such as 2,4-D. Many grass weed species
have been reported to develop resistance to this group of herbicides.

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